Justin Courcelle

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JoVE Journal Publications

ArticleTotal : 1	Year
Publication title Cited by 2	2010

Other Publications

Article	Year
Answering the Call: Coping with DNA Damage at the Most Inopportune Time. Journal of biomedicine & biotechnology\| PubMed ID: 12488586	2002
DNA damage-induced replication fork regression and processing in Escherichia coli. Science (New York, N.Y.)\| PubMed ID: 12543983	2003
RecA-dependent recovery of arrested DNA replication forks. Annual review of genetics\| PubMed ID: 14616075	2003
RecO acts with RecF and RecR to protect and maintain replication forks blocked by UV-induced DNA damage in Escherichia coli. The Journal of biological chemistry\| PubMed ID: 14625283	2004
RuvAB and RecG are not essential for the recovery of DNA synthesis following UV-induced DNA damage in Escherichia coli. Genetics\| PubMed ID: 15126385	2004
When replication travels on damaged templates: bumps and blocks in the road. Research in microbiology\| PubMed ID: 15142619	2004
Recs preventing wrecks. Mutation research\| PubMed ID: 16011837	2005
Nucleotide excision repair or polymerase V-mediated lesion bypass can act to restore UV-arrested replication forks in Escherichia coli. Journal of bacteriology\| PubMed ID: 16199565	2005
Nascent DNA processing by RecJ favors lesion repair over translesion synthesis at arrested replication forks in Escherichia coli. Proceedings of the National Academy of Sciences of the United States of America\| PubMed ID: 16754873	2006
Monitoring DNA replication following UV-induced damage in Escherichia coli. Methods in enzymology\| PubMed ID: 16793416	2006
RuvABC is required to resolve holliday junctions that accumulate following replication on damaged templates in Escherichia coli. The Journal of biological chemistry\| PubMed ID: 16895921	2006
Structural conservation of RecF and Rad50: implications for DNA recognition and RecF function. The EMBO journal\| PubMed ID: 17255941	2007
Inactivation of the DnaB helicase leads to the collapse and degradation of the replication fork: a comparison to UV-induced arrest. Journal of bacteriology\| PubMed ID: 17526695	2007
RecBCD and RecJ/RecQ initiate DNA degradation on distinct substrates in UV-irradiated Escherichia coli. Radiation research\| PubMed ID: 17903041	2007
RecA433 cells are defective in recF-mediated processing of disrupted replication forks but retain recBCD-mediated functions. Mutation research\| PubMed ID: 18782580	2008
Shifting replication between IInd, IIIrd, and IVth gears. Proceedings of the National Academy of Sciences of the United States of America\| PubMed ID: 19357302	2009
Nucleotide excision repair is a predominant mechanism for processing nitrofurazone-induced DNA damage in Escherichia coli. Journal of bacteriology\| PubMed ID: 19465649	2009
ATP binding, ATP hydrolysis, and protein dimerization are required for RecF to catalyze an early step in the processing and recovery of replication forks disrupted by DNA damage. Journal of molecular biology\| PubMed ID: 20558179	2010
Escherichia coli Fpg glycosylase is nonrendundant and required for the rapid global repair of oxidized purine and pyrimidine damage in vivo. Journal of molecular biology\| PubMed ID: 21601577	2011